Top Stories
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A newly discovered Alzheimer’s gene appears to drive the first appearance of amyloid plaque in the brain, and could lead to new therapies that prevent the disease from developing.
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Seizures are usually considered a side effect of brain cancer, but a new Columbia University study of mice suggests they may also fuel the further growth of brain tumors.
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A new study shows how modifications to the tau protein may influence the different ways it can misfold in the brain, which is closely linked to the type of neurodegenerative disease that develops.
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This July, Columbia neurologist Mitch Elkind will become president of the American Heart Association, only the second time a neurologist has led the organization.
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Latest News
- October 3, 2017
Scientists propose a new model for Alzheimer's disease that may explain the high failure rate of recent clinical trials.
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- September 18, 2017
A cellular defense against protein aggregates suppresses ALS in early stages but later hastens spread of the disease, a study in mice suggests.
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- August 29, 2017
Age-related memory loss was reversed by boosting blood levels of osteocalcin, a hormone produced by bone cells, in mice.
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- August 21, 2017
Memories "lost" to Alzheimer's disease may just be hard to retrieve, suggests a new study by neuroscientists at Columbia University Medical Center.
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- July 26, 2017
Columbia neurologist Denise Chou, MD, explains a new breakthrough in headache medicine: the first targeted preventive therapy for migraine.
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- July 17, 2017
A new study suggests how nutrients found in brain-healthy foods can reduce inflammation and lower Alzheimer's risk.
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- June 21, 2017
Researchers have found the first direct evidence that autoimmunity plays a role in Parkinson’s disease.
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- May 25, 2017
Certain women with preeclampsia are more likely to have a pregnancy-related stroke, finds a new study led by Columbia neurologists.
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- May 19, 2017
Neurosurgeon Sameer Sheth uses deep brain stimulation to treat depression in patients who have not been helped with other therapies.
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- May 15, 2017
Spinal muscular atrophy is partly due to defects in the sensory neuron synapses that activate motor neurons. Symptoms may be reduced by improving synapse function.
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